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UW Med Fndtn Centennial Bldg
1685 Highland Ave 4142
Madison WI 53705-2281
University of Chicago Pritzker School of Medicine, Chicago, Illinois – MD
University of Chicago, Chicago, Illinois – PhD in Pathology
University of Washington Medical Center, Seattle, Washington – Residency in Internal Medicine
University of Wisconsin Hospital and Clinics – Fellowship in Endocrinology, Diabetes, and Metabolism
- The prostaglandin E<sub>2</sub> EP3 receptor has disparate effects on islet insulin secretion and content in β-cells in a high-fat diet-induced mouse model of obesity
- What the BTBR/J mouse has taught us about diabetes and diabetic complications
- Metformin Monotherapy Alters the Human Plasma Lipidome Independent of Clinical Markers of Glycemic Control and Cardiovascular Disease Risk in a Type 2 Diabetes Clinical Cohort
- Plasma Prostaglandin E<sub>2</sub> Metabolite Levels Predict Type 2 Diabetes Status and One-Year Therapeutic Response Independent of Clinical Markers of Inflammation
- Pancreatic Stellate Cells Prolong Ex Vivo Islet Viability and Function and Improve Engraftment
- Cholecystokinin attenuates β-cell apoptosis in both mouse and human islets
- TCF19 Impacts a Network of Inflammatory and DNA Damage Response Genes in the Pancreatic β-Cell
- PREVENT: A Randomized, Placebo-controlled Crossover Trial of Avexitide for Treatment of Postbariatric Hypoglycemia
- Pyruvate Kinase Controls Signal Strength in the Insulin Secretory Pathway
- Intra-islet GLP-1, but not CCK, is necessary for β-cell function in mouse and human islets
- Imaging and therapy of diabetes: State of the art
- Roux en Y gastric bypass hypoglycemia resolves with gastric feeding or reversal: Confirming a non-pancreatic etiology
- Interleukin 6 protects pancreatic β cells from apoptosis by stimulation of autophagy
- Successful in vitro fertilization and generation of transgenics in Black and Tan Brachyury (BTBR) mice
- Glucagon-like peptide-1 and cholecystokinin production and signaling in the pancreatic islet as an adaptive response to obesity
Dr. Davis runs a federally funded research lab focused on pancreatic beta cell biology. Understanding the mechanisms underlying beta cell growth, survival and death may help identify new therapeutic targets that can help drive beta cell proliferation in the patient with prediabetes, type 2 diabetes, a recent islet transplant, or potentially even type 1 diabetes in combination with immune therapies. She also conducts collaborative clinical research that investigates biomarkers for diabetes, novel dietary interventions for weight loss and diabetes prevention, and the treatment of diabetes in hospitalized patients.
Dr. Dawn Davis is a tenured Professor in the Division of Endocrinology, Diabetes, and Metabolism within the Department of Medicine. She is also the Endocrinology Section Chief at the William S. Middleton Memorial VA Hospital. Dr. Davis is the Director of the UW-Madison Comprehensive Diabetes Center, which focuses on building research collaborations and opportunities across multiple schools and departments at UW-Madison. She has received a Puestow Research Award and a Dickie Research Award from the Department of Medicine, a UW Health Patient Experience Physician Champion Award, and a Junior Physician Investigator Award and Medical Research Scholar Award from the American Federation for Medical Research. She holds a Senior Clinician Scientist Investigator Award from the Department of Veterans Affairs for her distinguished service and research accomplishments in support of Veterans.